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Cover -- Contents -- Part I. Taking Genetics Seriously -- 1. Introduction -- 2. The Genetic Lottery -- 3. Cookbooks and College -- 4. Ancestry and Race -- 5. A Lottery of Life Chances -- 6. Random Assignment by Nature -- 7. The Mystery of How -- Part II. Taking Equality Seriously -- 8. Alternative Possible Worlds -- 9. Using Nature to Understand Nurture -- 10. Personal Responsibility -- 11. Difference without Hierarchy -- 12. Anti-Eugenic Science and Policy -- Acknowledgments -- Notes -- Index.

This study used a behavioral genetic design to test whether three measures of pubertal timing moderated peer influence on risk‐taking in a sample of 248 female adolescent twin pairs (Mage = 16.0,SD = 1.5) from the National Longitudinal Study of Adolescent Health. Peer influence was operationalized as the quasi‐causal association between girls' self‐reported risk‐taking and the risk‐taking reported by their friends. Girls with earlier ages at menarche and who perceived themselves as more developed than peers were more susceptible to peer influence on risk‐taking. However, age‐standardized ratings of body changes did not moderate peer influence. This study highlights distinctions between multiple measures of pubertal timing, using an innovative synthesis of genetically informative data and peer nomination data.

This study examines whether changes in impulse control and sensation‐seeking across adolescence and early adulthood reflect independent or interdependent developmental processes. Data are drawn from a national longitudinal study (N = 8,270; 49% female; 33% Black, 22% Hispanic, 45% non‐Black, non‐Hispanic). An autoregressive latent trajectory model is used to test whether development in one trait influences development in the other. Although levels of these traits are inversely correlated, we do not find evidence that change over time in either trait is influenced by the prior level of the other. This failure to reject the null hypothesis is consistent with the view that sensation‐seeking and impulse control are the products of distinct neuropsychological systems that develop independently of one another.

AbstractAttention‐Deficit Hyperactivity Disorder (ADHD) is a heterogeneous disorder that is highly impairing. Early, accurate diagnosis maximizes long‐term positive outcomes for youth with ADHD. Tests of executive functioning (EF) are potential tools for screening and differential diagnosis of ADHD subtypes. However, previous research has been inconsistent regarding the specificity and magnitude of EF deficits across ADHD subtypes. Here, we advance knowledge of the EF‐ADHD relationship by using: (1) dimensional latent factor models of ADHD that captures the heterogeneity of expression, and (2) a comprehensive, reliable battery of EF tasks and modeling relationships with a general factor of EF ability. We tested 1548 children and adolescents (ages 7–15 years) from the Texas Twin Project, a population‐based cohort with a diverse socioeconomic and ethnic composition. We show that EF deficits were specific to the inattention domain of ADHD. Moreover, we found that the association between EF task performance and inattention was stable across sociodemographic groups. Our results demonstrate that failures of executive control are selectively manifested as covert inattentive symptoms, such as trouble with organization, forgetfulness, and distractedness, rather than overt symptoms, such as inappropriate talkativeness and interruption. Future research, utilizing a bifactor characterization of ADHD in clinical samples, is needed to further refine understanding of the nature of cognitive deficits in ADHD across the full range of symptom variation.