Does Green Finance Induce Corporate Environmental Hypocrisy? Evidence from China's Green Credit Policy
In: FINANA-D-24-02118
10 Ergebnisse
Sortierung:
In: FINANA-D-24-02118
SSRN
In: Journal of business ethics: JBE, Band 191, Heft 4, S. 793-810
ISSN: 1573-0697
In: Emerging markets, finance and trade: EMFT, Band 59, Heft 8, S. 2383-2397
ISSN: 1558-0938
In: Emerging markets, finance and trade: EMFT, Band 59, Heft 7, S. 2056-2078
ISSN: 1558-0938
SSRN
In: Environmental science and pollution research: ESPR, Band 29, Heft 38, S. 57656-57668
ISSN: 1614-7499
In: Reproductive sciences: RS : the official journal of the Society for Reproductive Investigation, Band 26, Heft 2, S. 278-288
ISSN: 1933-7205
In: Ecotoxicology and environmental safety: EES ; official journal of the International Society of Ecotoxicology and Environmental safety, Band 207, S. 111306
ISSN: 1090-2414
In: Reproductive sciences: RS : the official journal of the Society for Reproductive Investigation, Band 27, Heft 3, S. 895-904
ISSN: 1933-7205
Lipoprotein(a) (Lp(a)) is an important causal cardiovascular risk factor, with serum Lp(a) levels predicting atherosclerotic heart disease and genetic determinants of Lp(a) levels showing association with myocardial infarction. Lp(a) levels vary widely between populations, with African-derived populations having nearly 2-fold higher Lp(a) levels than European Americans. We investigated the genetic basis of this difference in 4464 African Americans from the Jackson Heart Study (JHS) using a panel of up to 1447 ancestry informative markers, allowing us to accurately estimate the African ancestry proportion of each individual at each position in the genome. In an unbiased genome-wide admixture scan for frequency-differentiated genetic determinants of Lp(a) level, we found a convincing peak (LOD = 13.6) at 6q25.3, which spans the LPA locus. Dense fine-mapping of the LPA locus identified a number of strongly associated, common biallelic SNPs, a subset of which can account for up to 7% of the variation in Lp(a) level, as well as >70% of the African-European population differences in Lp(a) level. We replicated the association of the most strongly associated SNP, rs9457951 (p = 6 × 10(-22), 27% change in Lp(a) per allele, ∼5% of Lp(a) variance explained in JHS), in 1,726 African Americans from the Dallas Heart Study and found an even stronger association after adjustment for the kringle(IV) repeat copy number. Despite the strong association with Lp(a) levels, we find no association of any LPA SNP with incident coronary heart disease in 3,225 African Americans from the Atherosclerosis Risk in Communities Study.
BASE