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Asthma is a chronic inflammation disease in airways which is emit bronchus hyperactivity. Asthma is a worldwide trouble that influence at least 1-18% population in the entire world. Exercises is a non-pharmacological therapy which is can be applied for asthma case. Exercise that is applied on this research is asthma gymnastic and breathing exercise. The purpose from this research is to get the effect of asthma gymnastic and breathing exercise on asthma status for asthma survivor.The research method use eksperiment in madupahat community in Semarang city with involve 19 persons, sample was taken with purposive sampling. The result of this research shows there is improvement in control status and the spirometri was increase in asthma survivors. The conclution in his research is asthma gymnastic and breathing exercise effective to control asthma status in asthma survivor.

Asthma is a chronic relapsing airways disease that represents a major public health problem worldwide. Intermittent exacerbations are provoked by airway mucosal exposure to pro-inflammatory stimuli, with RNA viral infections or inhaled allergens representing the two most common precipitants. In this setting, inducible signaling pathways the airway mucosa play a central role in the initiation of airway inflammation through production of antimicrobial peptides (defensins), cytokines, chemokines and arachidonic acid metabolites that coordinate the complex processes of vascular permeability, cellular recruitment, mucous hyper-secretion, bronchial constriction and tissue remodeling. These signals also are responsible for leukocytic infiltration into the submucosa, T helper-lymphocyte skewing, and allergic sensitization. Currently, it is well appreciated that asthma is a heterogeneous in terms of onset, exacerbants, severity, and treatment response. Current asthma classification methods are largely descriptive and focus on a single aspect or dimension of the disease. An active area of investigation on how to collect, use and visualize multidimensional profiling in asthma. This book will overview multidimensional profiling strategies and visualization approaches for phenotyping asthma. As an outcome, this work will facilitate the understanding of disease etiology, prognosis and/or therapeutic intervention.

Nearly 17% of people without asthma smoke. But surprisingly, even more people with asthma smoke. About 21% of people with asthma smoke, even though cigarette smoke is known to trigger asthma attacks. Smoking rates are different from state to state. The percentage of people with asthma who smoke ranges from about 12% in Minnesota and Utah to about 32% in Kentucky. The states with the highest percentage of people with asthma who smoke are clustered in the Midwest and South regions. See the map above for details. ; 2/1/13- date from document propertes ; CDC's National Asthma Control Program (NACP) was created in 1999 to help the millions of people with asthma in the United States gain control over their disease. The NACP conducts national asthma surveillance and funds states to help them improve their asthma surveillance and to focus efforts TN and resources where they are needed. ; Available via the World Wide Web as an Acrobat .pdf file (335.63 KB, 1 p.).

ObjectiveWe investigate the association between adult asthma and wealth, testing whether the disease impairs wealth accumulation (social selection model) or if wealth protects against asthma (social causation model).MethodsWe use the National Longitudinal Survey of Youth (n = 7,644) and linear and logistic regressions to estimate the association between wealth and asthma. Changes in relative wealth following an asthma diagnosis and asthma status by increases in wealth through inheritance provide evidence on the causal direction.ResultsAsthma, particularly severe asthma, is associated with lower wealth. Wealth ranking does not change after a diagnosis of asthma, but inheriting a substantial sum is associated with a lower risk of severe asthma.ConclusionWealth appears to protect against severe asthma, supporting the social causation model of disease.