Taxes and economic growth: an empirical analysis of Pakistan
In: European Law Review, Volume 8, Issue 6, p. 01-01
ISSN: 0307-5400
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In: European Law Review, Volume 8, Issue 6, p. 01-01
ISSN: 0307-5400
In: Global social sciences review: an open access, triple-blind peer review, multidisciplinary journal, Volume V, Issue II, p. 334-340
ISSN: 2616-793X
This paper is an effort to de-construct the two opposite ideas of hope and despair in Ahmed Ali's Twilight in Delhi and Khushwant Singh's Delhi. The comparative points of view regarding Delhi by Singh and Ali serve to construct the ideological, political and ontological framing of Delhi. The paper explores the significance of Delhi as a symbol of political energy which distributes power or snatches it as an active agent of power-history. The paper exhibits the socio-political, economic and communal structures portrayed by Ali and Singh. The study is an effort to detect the system of representation in the metaphor of Delhi in the context of power-resistance and failure of the struggle against the Raj. The paper tries to show that both Ali and Singh see Delhi with a difference of outlook and literary approach to manifest hope and despair.
In: Environmental science and pollution research: ESPR, Volume 30, Issue 56, p. 119134-119150
ISSN: 1614-7499
25 páginas, 6 figuras, 2 tablas ; Characterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE ε4 allele. ; This work was funded by a grant (EADB) from the EU Joint Programme – Neurodegenerative Disease Research. INSERM UMR1167 is also funded by the INSERM, Institut Pasteur de Lille, Lille Métropole Communauté Urbaine and French government's LABEX DISTALZ program (development of innovative strategies for a transdisciplinary approach to AD). Full consortium acknowledgements and funding are in the Supplementary Not ; Peer reviewed
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